The delayed senescence of postharvest buds in salt ions was related to antioxidant activity, HDA9 and CCX1 in broccoli (Brassica oleracea L. var. Italic Planch.)

文献类型： 外文期刊

第一作者： Yan, Kai

作者： Yan, Kai;Ran, Maolin;Li, Shengnan;Zhang, Junli;Wang, Yu;Wang, Zhimin;Wei, Dayong;Tang, Qinglin;Yan, Kai;Yan, Kai;Li, Shengnan;Zhang, Junli;Wang, Yu;Wang, Zhimin;Wei, Dayong;Tang, Qinglin;Ran, Maolin

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关键词： Broccoli; Salt ions; Senescence; CCX1; HDA9

期刊名称：FOOD CHEMISTRY （ 影响因子：7.514； 五年影响因子：7.516 ）

ISSN： 0308-8146

年卷期： 2020 年 324 卷

页码：

收录情况： SCI

摘要： Epigenetic regulation and salt ions play essential roles in senescence control, but the underlying regulatory mechanism of senescence has not been thoroughly revealed in broccoli postharvest buds. Here, we found 200 mmol.L-1 NaCl, 400 mmol.L-1 KCl, 40 mmol.L-1 CaCl2 and 0.5 mu mol.L-1 Trichostatin-A (TSA, a histone deacetylase inhibitor) delayed the bud senescence. They resulted in significantly inhibiting the malondialdehyde (MDA) content, and dramatically promoting the contents of superoxide dismutase (SOD), peroxidase (POD) and Chlorophyll. Furthermore, the expression of PHEOPHYTINASE (PPH) and NONYELLOWING (NYE1), but not SUPPRESSOR OF OVEREXPRESSION OF CONSTANS 1 (SOC1), were remarkably repressed by salt ions and TSA. Interestingly, HISTONE DEACETYLASE 9 (HDA9) and CATION/Ca2+ EXCHANGER 1 (CCX1) were down-regulated by NaCl, CaCl2 and TSA. Further assays demonstrated that HDA9 could not interact with CCX1 promoter. It suggested that CCX1 along with HDA9 were involved in inhibiting the senescence of broccoli buds, and regulated aging by indirect interaction.

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